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Emerging-market Multinational Enterprises in East Central Europe [1 ed.] ,

Table of contents :
Preface
Acknowledgements
Contents
Notes on Contributors
List of Figures
List of Tables
Part I: Introduction
Chapter 1: Theories of Internationalization and Foreign Direct Investment: How to Explain FDI from Emerging MNEs?
Introduction
A Brief Overview of Traditional and New Theories
Traditional Theories
The Japanese School of FDI
New Theoretical Attempts
Driving Forces and Location Choices Behind the International Expansion Strategy of Emerging MNEs: Push and Pull Factors
The Book’s objectives
References
Chapter 2: Changing Trends of Foreign Direct Investments in East Central Europe
Introduction: Different Worlds
FDI in the Process of ECE Transition
FDI and GDP in Figures
ECE Countries in the Global Labor Division
Potential Future Scenarios
References
Part II: Emerging Multinationals from Asia
Chapter 3: Home and Host Country Determinants of Chinese Multinational Enterprises’ Investments into East Central Europe
Introduction
Driving Forces Behind the International Expansion Strategies of Chinese MNEs
Characteristics of Chinese FDI Globally
Push Factors and Public Policies Behind Chinese Outward FDI
Chinese Outward FDI in ECE
Characteristics and Major

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Abstract
Sensory abnormalities are commonly recognized as diagnostic criteria in autism spectrum disorder (ASD), as reported in the last edition of the Diagnostic and Statistical Manual of Mental Disorder (DSM-V). About 90% of ASD individuals have atypical sensory experiences, described as both hyper- and hypo-reactivity, with abnormal responses to tactile stimulation representing a very frequent finding. In this review, we will address the neurobiological bases of sensory processing in ASD, with a specific focus of tactile sensitivity. In the first part, we will review the most relevant sensory abnormalities detected in ASD, and then focus on tactile processing deficits through the discussion of recent clinical and experimental studies. In the search for the neurobiological bases of ASD, several mouse models have been generated with knockout and humanized knockin mutations in many ASD-associated genes. Here, we will therefore give a brief overview of the anatomical structure of the mouse somatosensory system, and describe the somatosensory abnormalities so far reported in different mouse models of ASD. Understanding the neurobiological bases of sensory processing in ASD mouse models may represent an opportunity for a better comprehension of the mechanisms underlying sensory
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PERSIAN AUTHORS Elect ASIA Lesser PART 1
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